From: Reported concepts for the treatment modalities and pain management of temporomandibular disorders
Name of the theory | Statements of the theory |
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Mechanical displacement (by Costen) | Lack of support in lateral teeth or functional occlusal premature contacts lead to direct eccentric positioning of the condyle in the glenoid fossa; this leads to pain, ear symptoms, adverse muscle activity and TMD |
Trauma theory (by Zack and Speck) | The principal factor of TMD is micro-/macro-trauma; trauma can cause structural alternation to the muscles or directly to the joint structures |
Biomedical (by Reade) | Disorder is initiated by trauma; specific factors (malocclusion, parafunctions, occupational activities) cause the progression of the symptoms |
Osteoarthric (by Stegenga) | Osteoarthrosis is a main cause of TMD; muscular symptoms and systemic diseases are secondary to TMJ pathology |
Muscle (by Travell and Rinzler) | Masticatory muscles are the primary etiologic factor to TMD; myalgia (caused by chronic myospasm) is secondary to parafunctions and can refer pain to TMJ |
Neuromuscular (by Ramfjord) | Occlusal problems cause TMDs, the loss of occlusal equilibrium leads to the incoordination of muscles and spasms |
Psychophysiological (by Schwartz and Laskin) | TMD occurs outside of the physical factors; psychosocial factors play a crucial role in TMD pathogenesis – the main factor of hypertension and overcontraction of the muscle is due to the parafunctions performed to relieve stress |
Psychosocial theory (by Dworkin) | Emotional disturbances induce hyperactivity of the muscles and lead to parafunctional habits and occlusal anomalies; the muscle contractivity is accentuated with teeth clenching, and repeatability leads to pain |