Influences of smoking and caffeine consumption on trigeminal pain processing

Table 2 Results of the pain-related evoked potentials before and after consuming caffeine 400 mg or smoking a cigarette

Groups	N2	N2	P2	P2	*PPA*	PPA
	*Before*	*After*	*Before*	*After*	*Before*	*After*
Coffeine	182.60 ± 12.49	182.43 ± 11.24	131.78 ± 10.39	131.78 ± 9.60	52.03 ± 20.46	48.48 ± 22.64
	[165.90-211.70]	[161.35-206.70]	[108.90-52.80]	[115.00-153.00]	[19.70-128.30]	[16.45-144.70
Nicotine	182.15 ± 12.49	176.89 ± 10.36***	131.35 ± 9.35	128.32 ± 7.93*	51.55 ± 26.32	46.07 ± 22.63
	[158.30-205.40]	[161.30-199.10]	[155.75-150.40]	[113.00-141.70]	[10.40-117.30]	[13.70-84.45]

Results are presented as mean ± standard deviations and range.
*p < 0.05 t-test paired samples.
**p < 0.005 t-test paired samples.
After smoking N2 and P2 latency are significantly reduced showing a facilitation of trigeminal pain processing at supraspinal level.

ISSN: 1129-2377